This is a summary of Chapter XII of “The Sensitive Nervous System” by David Butler. Intro Today we will take a look at assessing upper limb neurodynamic tests (ULNT). These assessments used to be called tension tests, but that terminology is now a defunct mechanical description. We now describe these as neurodynamic tests to better appreciate the neurophysiologic aspects of mechanosensitivity and upper limb homunculi stability. These tests are numbered based on the movement sensitizer, which are as follows: 1 – Shoulder abduction. 2 – Shoulder depression. 3 – Elbow flexion. ULNT1: Median Nerve Here is the quick test first. Here is how to do the manual test. A quick heads up regarding head motions. Sidebending away increases symptoms in 90% of people. Sidebending toward decreases symptoms in 70% of people. ULNT2: Median Nerve Here is the manual test ULNT2: Radial Nerve Here is the active test. And the manual test. ULNT3: Ulnar Nerve Here is the active test And the manual test. Musculocutaneous Nerve Here is the active test And the passive test. Axillary Nerve Here is the passive test. Suprascapular Nerve Here is the test. Final Words Have some fun with these tests, and be mindful that you are not too aggressive. Thanks to Scott and Sarah for your videotaping help. You guys rock.
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The Sensitive Nervous System Chapter V: Neurodynamics
This is a summary of Chapter V of “The Sensitive Nervous System” by David Butler. Intro Neurodynamics is the study and relationship of nervous system mechanics and physiology. The testing protocols for neurodynamics assess the nervous system’s ability to lengthen, glide, and change amongst interfacing structures. When discussing neurodynamics, it is important to think of the nervous system as a continuum. Mechanical, electrical, and chemical changes in one part of the nervous system affect other related parts. Gross Movements and Dynamics When having a nervous system, the following qualities, movements, and buffering capabilities are necessary: Slide, glide, strain. Elongate (think gymnasts) and return from elongated position. Compress (ulnar nerve during elbow flexion). Stength (kicking a field goal). Jolting (whiplash). Repetitive forces Bending Fluid/chemical selectivity. Neural Connective Tissue These include the meninges, nerve root complex, and peripheral nerve structures. Broken down as follows: Meninges Dura mater (outer, tougher) Arachnoid mater Pia mater (inner, thinner) Nerve root complex Root Sleeve Dorsal and ventral roots DRG Spinal nerve. Peripheral nerves Epineurium Perineurium Endoneurium Mesoneurium – Sheath that surrounds a nerve. Contracts like an accordion to glide along adjacent tissues. Can become fibrotic with injury. Important Attachments Meningovertebral ligaments – anchor down to spinal canal, which could become symptomatic. Rectus capitus posterior is connected to the dura mater between the occiput and atlas; helping the dura fold. Makes you wonder what you are truly doing when you release this structure. The sympathetic trunk’s proximity to the spinal column makes it susceptible to increased loads
Read MoreThe Sensitive Nervous System Chapter X: Neurodynamic Tests in the Clinic
This is a summary of Chapter X of “The Sensitive Nervous System” by David Butler. The Tests When assessing neurodynamics, there is a general system that is used including the following tests: Passive neck flexion (PNF). Straight leg raise (SLR). Prone knee bend (PKB). Slump. 4 different upper limb neurodynamic tests (ULNT). I will demonstrate these tests for you in later chapters. Many clinicians when discussing the lower extremity-biased tests deem that maybe only one or two of the tests need to be performed, however this assertion is erroneous. Slump, SLR, and PNF all need to be tested as a cluster. The reason being is that the clinical responses may often differ. This difference is especially noticeable when comparing the SLR and the slump. These two are not equal tests for the following reasons: Components are performed in a different order. Spine position is different. Patients may be more familiar with the SLR, therefore give more familiar responses. The patient is in control during the slump, not in the SLR. The slump is more provocative. Rules of Thumb When testing neurodynamics, here are the following guidelines: 1) Active before passive. 2) Differentiate structures – add/subtract other movements to see if symptoms can change. 3) Document the test order. Positive Test The positive testing here is a little dated based on what Butler’s group and the research says as of right now. Based on what I have learned from Adriaan Louw, having any of the following is what constitutes a positive
Read MoreThe Sensitive Nervous System Chapter XIV: Management Strategies: Integration of Neurodynamics
This is a summary of chapter XIV of “The Sensitive Nervous System” by David Butler. The Big Picture Evidence Based Approach Here is the step by step patient care process that Butler advocates. 1) Identify red flags and manage accordingly. 2) Educate on the whole problem to include tissue health status, the nervous system’s role, and test results. 3) Provide prognosis and make realistic goals. 4) Promote self-care, control, and motivation. 5) Decrease unnecessary fear and manage catastrophization. 6) Get patients moving as early as possible. 7) Help patients identify success and sense of mastery of a problem. 8) Perform a skilled exam. 9) Acknowledge that biopsychosocial inputs combine with the nervous system to produce pain and disability. 10) Use any measures possible to reduce pain. 11) Minimize number of treatments and contacts with all medical personnel. 12) Chronic pain may need a multidisciplinary approach. 13) Manage physical function and dysfunction. 14) Assess and assist in improving general fitness. 15) Assess how injury affects creative outlets and assist the patient with regaining creativity and discovering new creative outlets. Incorporating Neurodynamics There are several ways to incorporate neurodynamics into the patient’s plan of care which will be outlined below. Reassessment. Explanation. Passive mobilization. Active mobilization. Posture and ergonomics. Reassessment There are many evaluation protocols that warrant constant reassessment after applying an intervention. Be it a comparable sign or audit, neurodynamic tests can be utilized well within these systems. A word of caution with instant reassessment, as quick changes could merely be
Read MoreOnline Consult with The Manual Therapist
The Rundown My good friend Erson Religioso of The Manual Therapist fame recently contacted me to do a consult for some back/leg trouble he has been having. It was a very interesting eval for many reasons. Online consults are a completely different animal, as you cannot do any hands-on testing. Moreover, when you have a therapist who is initiated into pain neuroscience, you don’t have to go so much the Explain Pain route 🙂 So with this eval, we looked at things a lot through a PRI lens, and were able to get him strategies to modulate his pain experience. The eval runs a smidge over 1 hour, so here are some vids with a quick rundown. Subjective – Getting paresthesia down the R LE that began 2 weeks ago after a car ride…has peripheralized since initial event. – Symptoms are aggravated with static sitting or standing…onset ranging from seconds to minutes. – Has tried loading/unloading MDT strategies, neurodynamics, Mulligan techniques, IASTM, compression wrapping, etc…all to no avail. Objective (major findings) – Limited B Apley’s scratch (1 per FMS scoring) – Negative slump and ASLR – Painful lumbar motions of extension, right rotation and sidebend. R sidebend was limited. – Negative thomas test on left, positive on right – Slight limitations in active seated hip IR B, R>L. – Adduction lift scores 1/5 B. My Impression If I were to classify Erson, it seems his symptoms would seems to be more dominant as peripheral nociceptive ischemic and central sensitivity (he stated he has
Read MoreThe Sensitive Nervous System Chapter XI: Neurodynamic Testing for the Spine and Lower Limb
This is a summary of Chapter XI of “The Sensitive Nervous System” by David Butler. Intro For today’s chapter, I have decided that the best way to learn these tests is to show you. I will write in any pertinent details you need for a good test performance. The Straight Leg Raise (SLR) SLR hacks. Add sensitizers (dorsiflexion, plantarflexion, etc) to determine nervous system involvement. Add cervical flexion or visual input to enhance responses. Be mindful of symptoms before and after pain responses. If this test is positive post-operation, it will likely be inflammatory in nature. You can preload the system further with cervical flexion or sidebending the trunk away from the test side. Here are some other ways to perform the SLR with sensitizers first. (I apologize for the way the camera shot in advance). For tibial nerve-bias. For fibular nerve bias. For sural nerve bias. Passive Neck Flexion (PNF) Here is how to perform the test. PNF Hacks. Add SLR to further bias the test. Be mindful of Lhermitte’s sign, which is an electric shock down the arms or spine. This is a must-refer sign as there is potential spinal cord damage. Slump Test Here is how to perform the slump. Slump Knee Bend In the book itself, Butler uses the prone knee bend as his base test. However, NOI does not teach this motion as much and now favors the slump knee bend. This movement allows for much more differentiation to be had. And the saphenous nerve
Read MoreThe Sensitive Nervous System Chapter I: Painting a Bigger Canvas
This is a summary of Chapter I of The Sensitive Nervous System. This book is an all-encompassing manual regarding neurodynamics. This concept is defined as the physical and related physiological abilities of the nervous system. Before delving into neurodynamic nitty-gritty, a brief history of physical therapy is laid out via a very cool brachial plexus design (you have to get the book to see it). There are three different progressions in physical therapy history: manual therapy, exercise, and neurological manual therapy. The first time PTs learned manipulation was in 1916 at St. Thomas Hospital in London. The thought process of the time, as well as most early manual therapy, was predominantly biomechanically joint-centric. Eventually, muscle and other tissues were targeted. These approaches were championed by Geoffrey Maitland’s signs and symptoms approach and Graves’ pathological model. Concomitant with manual therapy has been exercise, which had moved from nonspecific (aerobics, tai chi) to specific movements a la Vladimir Janda and Shirley Sahrmann. On the other side of orthopedic manual therapy were manual techniques from the likes of Bobath’s NDT and PNF. What is sad about these techniques is that they have not interacted much during manual therapy’s development. Butler makes arguably one of the most important statements in the book by saying our patients are ultimately all neurological. We will all meet at the brain. Aside from various manual approaches, recent techniques have been developed including psychology, counseling, exercise physiology, and acupuncture. Butler feels these are nice adjuncts to the plan of
Read MoreBook Notes
“Clinical Neurodynamics” by Michael Shacklock Chapter 1: General Neurodynamics Chapter 2: Specific Neurodynamics Chapter 3: General Neuropathodynamics Chapter 4: Diagnosis of Specific Dysfunctions Chapter 5: Diagnosis with Neurodynamic Tests Chapter 6: Planning the Physical Examination Chapter 7: Standard Neurodynamic Testing Chapter 8: Method of Treatment: Systematic Progression Chapter 9: Cervical Spine Chapter 10: Upper Limb Chapter 11: Lumbar Spine Chapter 12: Lower Limb “Explain Pain” By David Butler & Lorimer Moseley Section 1: Intro to Pain Section 2: The Alarm System Section 3: The Damaged and Deconditioned Body Section 4: Altered Central Nervous System Alarms Section 5: Modern Management Models Section 6: Management “Movement” by Gray Cook Chapter 1: Introduction to Screening and Assessment Chapter 2: Anatomical Science vs. Functional Science Chapter 3: Understanding Movement Chapter 4: Movement Screening Chapter 5: Functional Movement Systems and Movement Patterns Chapter 6: Functional Movement Screen Descriptions Chapter 7: SFMA Introduction and Top Tier Tests Chapter 8: SFMA Assessment Breakout Descriptions and Flowcharts Chapter 9: Analyzing the Movements in Screens and Assessments Chapter 10: Understanding Corrective Strategies Chapter 11: Developing Corrective Strategies Chapter 12: Building the Corrective Framework Chapter 13: Movement Pattern Corrections Chapter 14: Advanced Corrective Strategies Chapter 15: In Conclusion “Recognizing and Treating Breathing Pattern Disorders” by Leon Chaitow Chapter 1: What are Breathing Pattern Disorders? Chapter 1: The Structure and Function of Breathing Chapter 2: Patterns of Breathing Function in Hyperventilation Syndrome and Breathing Pattern Disorders Chapter 3: Biochemical Aspects of Breathing Chapter 4: Biomechanical Influences on Breathing “The Sensitive Nervous System”
The Sensitive Nervous System Chapter VII: Assessment with a Place for the Nervous System
This is a summary of Chapter VII of “The Sensitive Nervous System” by David Butler. Education When it comes to patient education, there are four things that every patient wants to know: 1) What is wrong with me? 2) How long will it take to get better? 3) What can I do for it? 4) What can you (the clinician) do for it? When we do educate, we must not forget that pain is a biopsychosocial phenomenon and multifactorial. The onion skin model below provides a good relationship analogy for this. The first goal addressed in education is making the patient understand pain. Patients must realize that pain is the defender, not the offender. It is our body’s way to perceive a threat. Therefore, we must quell this fear before focusing on function. Here are some suggested ways to describe pain in non-threatening ways. Back trouble. Neck discomfort. Twinges. Feelings. When obtaining pain information from our patients, this is something that we do not have to measure. Instead, it is important to look at variables associated with pain, namely. 1) Geography & nature, aggravating/relieving factors, links. 2) Mechanism of injury. 3) Explore how patient’s classify their symptoms (e.g. my joints are worn out), and ask why they think the symptoms still persist. 4) Consequences of the pain. 5) Coping types. 6) How the patient relates to pain (do they get angry or play the blame game). When determining treatment course, instead of focusing on the structure at fault, look at
Read MoreThe Sensitive Nervous System Chapter III: Pain Mechanisms and Peripheral Sensitivity
This is a summary of Chapter III of “The Sensitive Nervous System” by David Butler. Intro When we discuss peripheral issues, we are not only talking about the pathoanatomical source, but pathobiological processes dominating the clinical picture. There are several instances in which the pathoanatomical model falls short: Phantom limb pain. Why pain persists post-healing. Why similar injuries heal faster in certain people. Why 10-14% of the world’s population have an ongoing pain state. Tissues do get injured, but we must not forget the nervous system’s intricate link to injury. When tissues are hurt, they repair but are unlikely to ever be the same again. To protect against further threat, the CNS has the ability to increase nerve sensitivity. This change happens only if the person decides consciously or subconsciously that there is a need for it, and does not occur in everyone. There are two ways in which this sensitivity develops; Primary sensitivity: Increased sensitivity to input at the injury site. Secondary sensitivity: Increased sensitivity to uninjured tissues around the injury. All pain is neurogenic, operates in a continuum, and has many components. Nociception (NOC) NOC is tissue pain that occurs at a neuron’s end that is excited by mechanical, thermal, or chemical stimuli. It does not always match up with tissue health status. A normal nerve ending has a very high firing threshold, and nearly 1/3 will never fire. These are called silent nociceptors. Looking at a chemical process such as inflammation shows us how these nerves fire.
Read MoreMovement Chapter 8: SFMA Assessment Breakout Descriptions and Flowcharts
This is a chapter 8 summary of the book “Movement” by Gray Cook. What to Look For The SFMA breakouts are utilized to determine if one’s movement deficiencies have a mobility or stability origin. There are further possibilities in each of these categories. It Could Be a Mobility Problem There are two subsets of mobility problems that include tissue extensibility dysfunction (TED) and joint mobility dysfunction (JMD). From here, we can break it down even further in each subset. Here are some potential TEDs Active/passive muscle insufficiency Limited neurodynamics (they said neural tension; come on Gray!) Fascial tension Muscle shortening Hypertrophy Trigger points Scarring/fibrosis And here are some potential JMDs Osteoarthritis/arthrosis Single-joint muscle spasm/guarding Fusion Subluxation Adhesive capsulitis Dislocation It could be a Stability Problem These issues are also known as stability or motor control dysfunction (SMCD). Most conventional therapies would treat these complaints by strengthening the stabilizers, but this is problematic. When something works reflexively, how can we train something volitionally and expect changes? To train these muscles we must focus on proprioceptive and timing-based training. There are several examples of SMCD problems. Motor control dysfunction. High threshold strategy. Local muscle dysfunction/asymmetry. Mechanical breathing dysfunction. Prime mover or global muscle compensation behavior or asymmetry. Poor static stability, alignment, postural control, asymmetry, and structural integrity. Poor dynamic stability, alignment, postural control, asymmetry, and structural integrity. Relatedness Mobility and stability can influence one another. If I were to lose mobility at one segment, motor control can be distorted at nearby segments.
Read MoreCourse Notes: Graded Motor Imagery
I recently attended another great course through the NOI Group called “Graded Motor Imagery” (GMI) taught by Bob Johnson. These guys are the industry leaders in all things pain so please check them out. It was great connecting with Bob and learning what I think will be an excellent adjunct to what I am currently doing. So here is the run down on GMI. Overview GMI is a three-pronged sequential process of establishing early, nonpainful motor programming. Johnson calls this synaptic exercise to limit negative peripheral pain expression. GMI is a 3 step process: 1) Laterality reconstruction (Implicit Motor Imagery). 2) Motor imagery (Explicit Motor Imagery). 3) Mirror Therapy. The Neuromatrix Paradigm & Pain States Before delving into the neuromatrix, we first must define pain. Pain is a multiple system output or expression by an individual-specific pain neuromatrix that activates when the brain concludes that body tissues are in danger and action is required. The neuromatrix, like I talk about in this post here, is the nervous system’s coding space and network. It is first and foremost affected by genetics, sculpted by experience, and constantly evolving. It is the entity that makes us who we are—the self. The neurosignature, or neurotag, is an output’s representation in the brain. For example, regions in the brain will activate in response to produce the pain output. This sequence is the neurosignature. Some common activated areas when pain is expressed include both primary and secondary somatosensory cortices, insula cortex, anterior cingulgate cortex, thalamus, basal
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