This is a summary of section 6 of “Explain Pain” by David Butler and Lorimer Moseley. Management 101 The most important thing you can understand is that no one has the answer for all pains. Pain is entirely individualistic, hence requiring different answers. There are several strategies which one can undertake to triumph over pain. Tool 1: Education Knowing how pain works is one of the most important components to overcoming pain. Instead of no pain, no gain, the authors like to use “know pain, or no gain.” Understanding pain is essential for squashing fear of pain, which leads best toward the road to recovery. Here are some important concepts to be known about explaining pain. Anyone can understand pain physiology. Learning about pain physiology reduces pain’s threat value. Combining pain education with movement approaches will increase physical capacity, reduce pain, and improve quality of life. Tool 2: Hurt ≠ Harm It is important to understand that when someone feels pain it does not equate with damage. The same can be said with recurring pains. These pain types are often ways to prevent you from making the same mistake twice. If your brain sees similar cues that were present with a previous injury, the brain may make you experience pain as a way to check on you and make sure you are okay. Just because hurt does not mean harm does not mean you can get crazy though. Because the nervous system is trying to protect you, it will take
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Explain Pain Section 5: Modern Management Models
This is a summary of section 5 of “Explain Pain” by David Butler and Lorimer Moseley. So Many Clinicians There are several people who would like to help someone in pain, with each person offering a different explanation and solution for someone’s pain. Research has shown these conflicting explanations can often make things worse. The one who has the most power over pain is the person who is in pain. Here are some general guidelines for someone dealing with pain. Make sure any injury or disease which requires immediate medical attention is dealt with. All ongoing pain states require a medical examination. Make sure any prescribed help makes sense and adds to your understanding of the problem. Get all your questions answered. Avoid total dependence on any practitioner. Make sure your goals are understood by you and the clinician. The clinician’s ultimate job is to assist you in mastering your situation. Models of Engagement There are 5 interchangeable models which enable both the patient and the clinician to identify the processes underlying pain. The orchestra model – Pain is a multi-component process that manifests itself in the brain and goes through many pathways. There are many players involved in the pain experience, hence the orchestra, with the brain as the maestro. The Onion Skin Model – Helps describe all the factors that go into the pain experience; including nociception, attitudes and beliefs, suffering, pain escape behaviors, and social environment. Fear-based models – Fear of pain and reinjury are major forces
Read MoreExplain Pain Section 4: Altered Central Nervous System Alarms
This is a summary of section 4 of “Explain Pain” by David Butler and Lorimer Moseley. CNS Alarms While much of talk in rehab deals with tissue injury and tissue pain, realize that the brain always makes the final decision as to whether or not you should feel pain. No brain, no pain. This sentiment does not mean that pain is not real. All pain is real. However, pain is a construct that the brain creates in order to ensure your survival. Spinal Cord Alarms When an injury occurs and the DRG receives impulses from peripheral structures or the brain, the spinal cord neurons must adapt to better uptake all these signals. In essence, the DRG becomes better at sending danger messages up to the brain. This change leads to short term increases in sensitivity to excitatory chemicals. Those stimuli that didn’t hurt before now do (allodynia) and those that used to hurt now hurt more (hyperalgesia). In persistent pain, this change continues occurring to the point where neurons that do not carry danger messages start growing into space where danger messages are taking place. Now innocuous stimuli such as grazing the skin begin hurting. The pain may be normal, but the underlying processes become abnormal. When these spinal cord alarm systems become unhealthy, the brain no longer receives an accurate message of what is going on. The alarms become magnified and distorted. The brain is told there is more damage in the tissues than is actually present. What is good is
Read MoreExplain Pain Section 3: The Damaged and Deconditioned Body
This is a summary of section 2 of “Explain Pain” by David Butler and Lorimer Moseley. Tissue Injury 101 When a body is damaged, pain is often the best guide to promote optimal healing. Sometimes it is good for us to rest, other times it is better to move. A similar healing process occurs for all tissue injuries. First, inflammation floods the injured area with immune and rebuilding cells. This reason is why inflammation is a good thing in early injury stages. A scar forms once the inflammatory process is over. The tissue then remodels to attempt to become as good as the original. Blood supply and tissue requirements determine how fast the healing process occurs. For example, ligaments heal much slower than skin because the former has a lower blood supply than the latter. This may also be a reason why aerobic exercise may speed up the healing process. If present, pain usually diminishes as the tissues heal. However, pain may persist if the nervous system still feels under threat. Acid and Inflammation The alarm sensors described here constantly work and often get us to move. Movement keeps our system flushed. When we don’t move or a physical obstruction is present (e.g. sitting), acid and by-products build up in the body tissues. Oftentimes we will start to feel aches and pains when we stay in a prolonged position, which is our body’s way of saying “get up and move.” Much like the alarm system, inflammation is a primitive way for our
Read MoreExplain Pain Section 2: The Alarm System
This is a summary of section 2 of “Explain Pain” by David Butler and Lorimer Moseley. Alarm Signals Our body’s alarm system alerts us to danger or potential danger. This alarm system is composed of sensors throughout the body, the eyes, nose, and ears. It is these sensors that are our first line of defense against harm. If one sensor fails the others take over. Most of these sensors are located in the brain and respond to various stimuli. Some to mechanical movement, some to temperature change; the sensors in the brain particularly respond to chemical activity. What is important to know with sensors is that they have a very short life expectancy of a few days. This cycling means our body’s sensitivity is constantly changing. It is with these life cycles that there is hope for those with chronic pain. Moreover, the rate at which sensors are made is normally stable but can change very quickly in regards to a particular stimulus. So if we take for example one with persistent pain, the rate at which pain sensitivity occurs can be changed. Nociception We lack pain receptors in our bodies. Instead, the various tissues have special neurons that respond to different stimuli. These receptors are called nociceptors, which translates into “danger receptors.” Nociception is occurring all the time, but only sometimes will it end in pain. Nociception is neither necessary nor sufficient for pain. The sensors correspond to particular neurons. In order for these neurons to become excited and
Read MoreThe Sensitive Nervous System Chapter XV: Clinical Aspects of Neurodynamics
This is a summary of chapter XV of “The Sensitive Nervous System” by David Butler. Intro In this chapter we discuss many specific neurodynamic pathologies and implementing the nervous system into treatment approach. Conservative Nervous System Decompression Here is a general step-by-step approach to decreasing threat throughout the nervous system. 1) Decrease tissue sensitivity by removing relevant stimuli and decreasing CNS threshold. 2) Improve container tissue health. 3) Improve the nerve tract’s ability to absorb traction forces. 4) Assess and improve the nerve to container relationship. 5) Assess/modify any adverse ergonomic or environmental factors. Carpal Tunnel Syndrome Tests to perform. ULNT1 & reverse. ULNT2 (median) & reverse. Compression (can add ULNT). Phalens and reverse Phalens. Phalens + ULNT. Treatment There are several options to treat carpal tunnel syndrome. Mobilizing not only the median nerve, but radial and ulnar is beneficial because the nerves are closely connected. Movement is critical because nerve inflammation and swelling does not leave the carpal tunnel easily. This problem is because there are minimal lymphatic channels in the tunnel. Nerve Root Complex Nerve root issues often have corresponding postural adaptations. Cervical – forward head posture. Lumbar – Flat lumbar spine with knees flexed, positioned toward the injured sign. In acute instance, it may be okay to let the patient rest in these antalgic postures until AIGS settle. Other presentations indicative of nerve root complex pathology include numbness/tingling down the extremities. Other possibilities include coldness, shooting, tiredness. Pain rarely goes into the extremities. Double Crush Double crush
Read MoreThe Sensitive Nervous System Chapter XIV: Management Strategies: Integration of Neurodynamics
This is a summary of chapter XIV of “The Sensitive Nervous System” by David Butler. The Big Picture Evidence Based Approach Here is the step by step patient care process that Butler advocates. 1) Identify red flags and manage accordingly. 2) Educate on the whole problem to include tissue health status, the nervous system’s role, and test results. 3) Provide prognosis and make realistic goals. 4) Promote self-care, control, and motivation. 5) Decrease unnecessary fear and manage catastrophization. 6) Get patients moving as early as possible. 7) Help patients identify success and sense of mastery of a problem. 8) Perform a skilled exam. 9) Acknowledge that biopsychosocial inputs combine with the nervous system to produce pain and disability. 10) Use any measures possible to reduce pain. 11) Minimize number of treatments and contacts with all medical personnel. 12) Chronic pain may need a multidisciplinary approach. 13) Manage physical function and dysfunction. 14) Assess and assist in improving general fitness. 15) Assess how injury affects creative outlets and assist the patient with regaining creativity and discovering new creative outlets. Incorporating Neurodynamics There are several ways to incorporate neurodynamics into the patient’s plan of care which will be outlined below. Reassessment. Explanation. Passive mobilization. Active mobilization. Posture and ergonomics. Reassessment There are many evaluation protocols that warrant constant reassessment after applying an intervention. Be it a comparable sign or audit, neurodynamic tests can be utilized well within these systems. A word of caution with instant reassessment, as quick changes could merely be
Read MoreThe Sensitive Nervous System Chapter XIII: Research and Neurodynamics: Is Neurodynamics Worthy of Scientific Merit?
This is a summary of Chapter XIII of “The Sensitive Nervous System” by David Butler. Intro Research has demonstrated that often evidenced-based medicine is low on the list for why clinicians choose a particular treatment. From an ethical standpoint, it is important to consider evidence. This chapter is very short so I will just provide the highlights that I got from it. Appraising a New Theory or Approach There are six criteria that a new theory should be evaluated by: 1) Support from anatomical and physiological evidence. 2) Designed for a specific population. 3) Studies from peer-reviewed journals. 4) Include a well-designed randomized controlled trial or single experiment. 5) Present potential side effects. 6) Proponents discuss and are open to limitations. Agreement Here are some definitions of different ways research measures agreement. – Cohen’s Kappa: Measures nominal data reliability. >0.75 is excellent agreement. 0.40-0.75 is fair to good. <0.40 is poor. – Pearson product movement correlation: Measures interval/ratio data. – ICC: Measures continuous data. The closer to 1, the better. Validity There are also many different validity types defined throughout this chapter. The first two are proven through logic and have the least evidence support. – Construct Validity: Valid relative to a theoretical foundation. – Content Validity: Can I use this measure to make an inference? The next two are higher up on the evidence support hierarchy. – Convergent Validity: The test shows a correlation between two variables. – Discriminant Validity: The test shows a low correlation between two variables.
Read MoreThe Sensitive Nervous System Chapter XII: Upper Limb Neurodynamic Tests
This is a summary of Chapter XII of “The Sensitive Nervous System” by David Butler. Intro Today we will take a look at assessing upper limb neurodynamic tests (ULNT). These assessments used to be called tension tests, but that terminology is now a defunct mechanical description. We now describe these as neurodynamic tests to better appreciate the neurophysiologic aspects of mechanosensitivity and upper limb homunculi stability. These tests are numbered based on the movement sensitizer, which are as follows: 1 – Shoulder abduction. 2 – Shoulder depression. 3 – Elbow flexion. ULNT1: Median Nerve Here is the quick test first. Here is how to do the manual test. A quick heads up regarding head motions. Sidebending away increases symptoms in 90% of people. Sidebending toward decreases symptoms in 70% of people. ULNT2: Median Nerve Here is the manual test ULNT2: Radial Nerve Here is the active test. And the manual test. ULNT3: Ulnar Nerve Here is the active test And the manual test. Musculocutaneous Nerve Here is the active test And the passive test. Axillary Nerve Here is the passive test. Suprascapular Nerve Here is the test. Final Words Have some fun with these tests, and be mindful that you are not too aggressive. Thanks to Scott and Sarah for your videotaping help. You guys rock.
Read MoreCourse Notes: Graded Motor Imagery
I recently attended another great course through the NOI Group called “Graded Motor Imagery” (GMI) taught by Bob Johnson. These guys are the industry leaders in all things pain so please check them out. It was great connecting with Bob and learning what I think will be an excellent adjunct to what I am currently doing. So here is the run down on GMI. Overview GMI is a three-pronged sequential process of establishing early, nonpainful motor programming. Johnson calls this synaptic exercise to limit negative peripheral pain expression. GMI is a 3 step process: 1) Laterality reconstruction (Implicit Motor Imagery). 2) Motor imagery (Explicit Motor Imagery). 3) Mirror Therapy. The Neuromatrix Paradigm & Pain States Before delving into the neuromatrix, we first must define pain. Pain is a multiple system output or expression by an individual-specific pain neuromatrix that activates when the brain concludes that body tissues are in danger and action is required. The neuromatrix, like I talk about in this post here, is the nervous system’s coding space and network. It is first and foremost affected by genetics, sculpted by experience, and constantly evolving. It is the entity that makes us who we are—the self. The neurosignature, or neurotag, is an output’s representation in the brain. For example, regions in the brain will activate in response to produce the pain output. This sequence is the neurosignature. Some common activated areas when pain is expressed include both primary and secondary somatosensory cortices, insula cortex, anterior cingulgate cortex, thalamus, basal
Read MoreThe Sensitive Nervous System Chapter XI: Neurodynamic Testing for the Spine and Lower Limb
This is a summary of Chapter XI of “The Sensitive Nervous System” by David Butler. Intro For today’s chapter, I have decided that the best way to learn these tests is to show you. I will write in any pertinent details you need for a good test performance. The Straight Leg Raise (SLR) SLR hacks. Add sensitizers (dorsiflexion, plantarflexion, etc) to determine nervous system involvement. Add cervical flexion or visual input to enhance responses. Be mindful of symptoms before and after pain responses. If this test is positive post-operation, it will likely be inflammatory in nature. You can preload the system further with cervical flexion or sidebending the trunk away from the test side. Here are some other ways to perform the SLR with sensitizers first. (I apologize for the way the camera shot in advance). For tibial nerve-bias. For fibular nerve bias. For sural nerve bias. Passive Neck Flexion (PNF) Here is how to perform the test. PNF Hacks. Add SLR to further bias the test. Be mindful of Lhermitte’s sign, which is an electric shock down the arms or spine. This is a must-refer sign as there is potential spinal cord damage. Slump Test Here is how to perform the slump. Slump Knee Bend In the book itself, Butler uses the prone knee bend as his base test. However, NOI does not teach this motion as much and now favors the slump knee bend. This movement allows for much more differentiation to be had. And the saphenous nerve
Read MoreThe Sensitive Nervous System Chapter X: Neurodynamic Tests in the Clinic
This is a summary of Chapter X of “The Sensitive Nervous System” by David Butler. The Tests When assessing neurodynamics, there is a general system that is used including the following tests: Passive neck flexion (PNF). Straight leg raise (SLR). Prone knee bend (PKB). Slump. 4 different upper limb neurodynamic tests (ULNT). I will demonstrate these tests for you in later chapters. Many clinicians when discussing the lower extremity-biased tests deem that maybe only one or two of the tests need to be performed, however this assertion is erroneous. Slump, SLR, and PNF all need to be tested as a cluster. The reason being is that the clinical responses may often differ. This difference is especially noticeable when comparing the SLR and the slump. These two are not equal tests for the following reasons: Components are performed in a different order. Spine position is different. Patients may be more familiar with the SLR, therefore give more familiar responses. The patient is in control during the slump, not in the SLR. The slump is more provocative. Rules of Thumb When testing neurodynamics, here are the following guidelines: 1) Active before passive. 2) Differentiate structures – add/subtract other movements to see if symptoms can change. 3) Document the test order. Positive Test The positive testing here is a little dated based on what Butler’s group and the research says as of right now. Based on what I have learned from Adriaan Louw, having any of the following is what constitutes a positive
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